By Veljko Vlaisavljević; Marko Došen; Borut Kovačič
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Additional info for Embryo quality and pregnancy outcome in infertile patients with endometriosis
One hypothesis is that secretion of various cytokines by inflammatory cells into the peritoneal cavity leads to proliferation of implants and recruitment of capillaries. Oxidative stress may be another component of the inflammatory reaction. Thus, the immune system may play a role in determining who will develop endometriosis, as well as the extent and clinical manifestation of the disease. It is said that these women had higher rates of autoimmune inflammatory diseases, hypothyroidism, fibromyalgia, chronic fatigue syndrome, allergies and asthma, compared with the general female population so, it could provide support for the theory of altered immune system in women with endometriosis.
Mol med. 2007; 9(2):  Brosens IA. Endometriosis–a disease because it is characterized by bleeding. Am J Obstet Gynecol. 1997; 176,263-267.  Fauconnier A, Chapron C. Endometriosis and pelvic pain: epidemiological evidence of the relationship and implications. Hum Reprod Update. 2005; 11,595-606.  Cramer D W. Epidemiology of endometriosis in adolescents. In: Wilson EA (ed) Endometriosis. Alan Liss, (1987) NewYork pp5–8  Kuohung W, Jones GL, Vitonis AF etal. Characteristics of patients with endometriosis in the United States and the United Kingdom.
Other less commonly involved regions include the vagina, cervix, rectovaginal septum, cecum, ileum, inguinal canals, abdominal or perineal scars, urinary bladder, ureters, and umbilicus. Rarely, endometriosis has been reported in the breast, pancreas, liver, gallbladder, kidney, urethra, extremities, vertebrae, bone, peripheral nerves, lung, diaphragm, central nervous system, and even in the prostate. Endometriosis is multifocal in most patients. The disease is staged according to site and severity of involvements.
Embryo quality and pregnancy outcome in infertile patients with endometriosis by Veljko Vlaisavljević; Marko Došen; Borut Kovačič