By William George Morice MD, PhD, Thomas V. Colby MD (auth.), Joseph F. Tomashefski Jr. MD, Philip T. Cagle MD, Carol F. Farver MD, Armando E. Fraire MD (eds.)
Dail and Hammar’s Pulmonary Pathology has demonstrated itself because the definitive reference within the box. This 3rd variation is now a two-volume, complete colour textual content led by way of Dr. Tomashefksi, as Editor-in-Chief and Drs. Cagle, Farver, and Fraire as affiliate Editors. the recent editorial board has endured to construct upon the distinction Dail and Hammar accomplished within the past versions via reorganizing, increasing and considerably revising the textual content. This authoritative reference paintings has been completely up to date to hide newly famous entities and the newest advances in molecular diagnostic suggestions. Abundantly illustrated with greater than 2000 complete colour illustrations. This awesome contribution to pathology literature is a must have for the library of each surgical and pulmonary pathologist.
Dail and Hammar’s Pulmonary Pathology has set the normal for which all different pathology texts try to accomplish. From stellar stories of the second one Edition:
"When the 1st version of Pulmonary Pathology by means of David H. Dail and Samuel P. Hammar used to be released in 1988, it used to be transparent that it was once to develop into the reference booklet for pulmonary pathology. It was once tricky to conceive then larger booklet at the topic might be assembled. the second one variation of this encyclopedic paintings proved convincingly that development might happen. certainly, the recent revised variation is extra whole and higher illustrated…I reflect on this booklet to be an exceptional contribution to the pathology literature and a needs to within the library of surgical and pulmonary pathologists." -- Human Pathology
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Additional info for Dail and Hammar’s Pulmonary Pathology: Volume II: Neoplastic Lung Disease
There is a predilection for lymphatic routes and the inﬁ ﬁltrates extend along bronchovascular bundles from the hilum. Hilar lymph nodes are involved. This is from an autopsy in a patient who had initially presented with lymph node disease. antigens not often expressed by lymphocytes such as CD15 and the activation-associated antigen CD30. For this reason the cellular origin of these cells was unclear. Detailed molecular genetic and immunophenotypic analyses have revealed that in most cases of classic Hodgkin’s lymphoma (>95%) the HRS cells are derived from postgerminal center B cells, and in the remainder they are derived from T cells.
Lymphomatoid granulomatosis. (A) Typical radiologic ﬁndings ﬁ with nodules, some showing cavitation. (B) Gross appearance with bulging, ﬁsh ﬁ ﬂesh–like nodules replacing much of the lung parenchyma. Necrosis is not prominent in this case. 99 Some contend that these “T-cell LYG” cases may be undersampled typical LYG cases, and indeed the detection of EBV-positive B cells in LYG may require studying multiple tissue blocks. However, rare cases with histologic features of LYG and the unusual attributes noted above do occur.
This likely reﬂects ﬂ the varying cytology of EBVassociated lymphoproliferative diseases, as detailed in the section on PTLD. In some instances these cases may be categorized as HIV-associated polymorphic B-cell lymphoma (PTLD-like) by the WHO criteria, and in other cases it is best to consider the lesions unclassiﬁ ﬁable and to reﬂ ﬂect this in the diagnosis. One must always bear in mind that immunoblastic reactions, some of which may be EBV associated, can occur in the setting of HIV. Given this, and the poor prognosis associated with higher grade lymphomas in AIDS patients, it is critical that ancillary immunoperoxidase and molecular genetic studies be performed to conﬁrm ﬁ a diagnosis of lymphoma and exclude a ﬂorid ﬂ reactive process in cases where there is any uncertainty regarding the histologic diagnosis.
Dail and Hammar’s Pulmonary Pathology: Volume II: Neoplastic Lung Disease by William George Morice MD, PhD, Thomas V. Colby MD (auth.), Joseph F. Tomashefski Jr. MD, Philip T. Cagle MD, Carol F. Farver MD, Armando E. Fraire MD (eds.)