By Thierry Passeron, Jean-Paul Ortonne
The pores and skin is coloured by means of a mix of pigments, which shape a part of a posh and hugely regulated method. Pigmentary defects mostly current with dyschromia and will be as a result of genetic defects, systemic affliction, inflammatory methods, metabolic defects, infections, tumors, or poisonous or iatrogenic motives. This atlas is an intensive textual content written via key opinion leaders inside of dermatology, it has a complete layout that publications the reader during the epidemiology, pathophysiology, analysis, therapy, and differential prognosis of either universal and infrequent pigmentary problems. It illustrates the prognosis and popularity of pigmentary problems with quite a lot of images.
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Additional resources for Atlas of Pigmentary Disorders
The coalescence of the small macules leads to poorly defined borders. Flesh-colored and mildly keratotic papules of lichen amyloidosis. Flesh-colored papules in micronodular amyloidosis. Friction amyloidosis due to the chronic use of a massage glove. Note the predominance of the lesions on the left part of the upper back in right-handed patients. EPIDEMIOLOGY Cutaneous amyloidosis is seen throughout the world but is more common among Asians, Middle-Easterners and Latin Americans. Mostly in adults, but all ages may be affected.
EPIDEMIOLOGY More frequent in women than in men, with a ratio of 6:1. More common in Caucasians than in Asians and Blacks. Can be observed at all ages but the onset is usually during the second or third decade of life. EXTRACUTANEOUS SIGNS None. KEY REFERENCES HISTOPATHOLOGY PATHOPHYSIOLOGY Unknown. The role for infection with Borrelia burgdorferi has been suggested. CLINICAL DERMATOLOGICAL PRESENTATION Single or multiple, sharply demarcated, hyperpigmented, non-indurated patches. With time the hyperpigmentation may lighten, and the lesion becomes depressed with sharply defined (cliffdrop) borders.
Note that the hyperpigmentation is associated with an increased superficial vascularization. Localization is very suggestive: horizontally across the forehead, sparing the hairline. At the contrary of melasma, the lesions are limited only on the forehead. EPIDEMIOLOGY Only few cases have been reported, but it is probably not so rare and frequently misdiagnosed with melasma. Mostly observed in adults. Men and women are affected. EXTRACUTANEOUS SIGNS None. HISTOPATHOLOGY PATHOPHYSIOLOGY Chemical photosensitizers and excessive sun exposure have been suggested, but the pathophysiology remains poorly understood.
Atlas of Pigmentary Disorders by Thierry Passeron, Jean-Paul Ortonne