By Fariba Behbod, Gailen D. Marshall Jr. (auth.), Phillip L. Lieberman MD, Michael S. Blaiss MD (eds.)
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Figure 3-6. Hereditary angioneurotic edema (HAE) . A and B, Angioedema of the hands. (continued on next page) 23 The Complement System Fi~~ure 3-6. (collti11lted) C, Five-year-old girl with a moderate attack of angioedema. D, Small bowel barium radiograph during an acute episode of pain in the proband. Two segments of jejunum have mural edema: "thul1lbprinting" (thick arrows) and spiculation (thill arrows). This condition is one of several that are caused by deficiencies and dysfunction on complement regulator proteins.
Tryptase, a protease, has been used as a marker for severe anaphylactic reactions and remains in the circulation for a few hours. It has been used postmortem to establish anaphylaxis as a cause of death \22\. Acid hydrolases, oxidative enzymes, and chemotactic factors are also found in mast cells. The heavily sulfated proteoglycans heparin and chondroitin sulfate cause the metachromatic staining of human mast cells and basophils. These proteogylcans stabilize the proteases found in the secretory granules.
2. Gleich GJ: Mechanisms of eosinophil-associated inflammation . Allergy Ciill /111111111/(J/2000, 105:65 1-663 . Ellg/.! 3. Wardlaw AJ: Molecular ha sis for selective eosinophil tl"
Atlas of Allergic Diseases by Fariba Behbod, Gailen D. Marshall Jr. (auth.), Phillip L. Lieberman MD, Michael S. Blaiss MD (eds.)