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By J. Thomale, M. R. Müller, C. Buschfort, S. Seeber, M. F. Rajewsky (auth.), Prof. Dr. W. Hiddemann, Prof. Dr. T. Büchner, Prof. Doz. Dr. B. Wörmann, Prof. Dr. J. Ritter, Prof. Doz. Dr. U. Creutzig, M. Keating MD PhD, W. Plunkett MD PhD (eds.)

ISBN-10: 3642719600

ISBN-13: 9783642719608

ISBN-10: 3642719627

ISBN-13: 9783642719622

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Extra info for Acute Leukemias VII: Experimental Approaches and Novel Therapies

Sample text

Cancer Genet Cytogenet 70: 1 Terpstra W, Prins A, Ploemacher RE, Wognum BW, Wagemaker G, Lowenberg B, Wielenga JJ (1996) Long-term initiating capacity of a CD34sUbpopulation of acute myeloid leukemia. Blood 87: 2187-2194 16. Terstappen LWMM, Safford M, Konemann S, Loken MR, Zurlutter K, Biichner Th, Hiddemann W, Wormann B (1991) Flow cytometric characterization of acute myeloid leukemia. II. Extreme hetereogeneity at diagnosis. Leukemia 5,757-767 17. Van Lorn K, Hagemeijer A, Smit EME, LOwenberg B (1993) In situ hybridization on MayGriinwald, Giemsa-stained bone marrow and blood smears of patients with hematologic disorders allows detection of cell lineage-specific cytogenetic abnormalities.

2- 0- '" a; •

IDo 0"[ saga8L Fig. 1. Multiple mechanisms of DNA repair in mammalian cells. Primary DNA lesions are detectable by adduct-specific antibodies and can be measured by quantitative immuno-fluorescence analysis (lCA; shaded area); repair intermediates can be recognized by the comet assay (boxed area). The inhibition of distinct steps by three repair modifiers is indicated (06-benzylguanine inactivates the alkyltransferase AT; methoxyamine prevents DNA incision by AP-endonuclease(s); aphidicoline inhibits gap filling by DNA polymerases) profiles or to the therapeutic response.

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Acute Leukemias VII: Experimental Approaches and Novel Therapies by J. Thomale, M. R. Müller, C. Buschfort, S. Seeber, M. F. Rajewsky (auth.), Prof. Dr. W. Hiddemann, Prof. Dr. T. Büchner, Prof. Doz. Dr. B. Wörmann, Prof. Dr. J. Ritter, Prof. Doz. Dr. U. Creutzig, M. Keating MD PhD, W. Plunkett MD PhD (eds.)


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